Leading Scientists From The World Renowned Yale University Have Confirmed That COVID mRNA Vaccines Cause Vaccine Acquired Immune Deficiency Syndrome.
The Yale University School of Medicine found that mRNA injections alter human biology to create long term S2 spike protein production that increases over time.
The scientists warned that the COVID mRNA vaccines alter T-cell immunophenotypes which triggers VAIDS. The study was led by Bornali Bhattacharjee.
People not only collectively in essence felt forced to take an experimental vaccine or risk losing their jobs…we were lied to about it. It never stopped transmission, it never stopped contracting it & it was never safe.
The mRNA vaccines cause heart issues, blood clots, autoimmune disorders & so many more grave illnesses & death. This is the most egregious scandal in worldwide history. COVID-19 vaccines cause adverse events such as myocarditis & pericarditis, thrombosis & thrombocytopenia, Guillain–Barre syndrome, transverse myelitis & Bell’s Palsy
In addition, individuals have reported post-vaccination symptoms resembling long COVID beginning shortly after vaccination. This condition, sometimes referred to as post-vaccination syndrome (PVS) or post-acute COVID-19 vaccination syndrome (PACVS), is characterized by symptoms such as exercise intolerance, excessive fatigue, numbness, brain fog, neuropathy, insomnia, palpitations, myalgia, tinnitus, headache, burning sensations & dizziness.
Finally, we have scientific confirmation that vaccination against COVID-19 causes a marked decrease in immunity to heterologous pathogens such as viruses, bacteria & fungi.’ A condition known as ‘VAIDS’ vaccine-induced AIDS.” COVID mRNA vaccines cause long-term symptoms by multiple mechanisms:
#1 Vaccine 80 components, the mRNA lipid nanoparticles & adenoviral vectors, trigger activation of pattern recognition receptors. Thus, unregulated stimulation of innate immunity leads to chronic inflammation.
#2 The S2 spike protein expressed following BNT162b2 or mRNA-1273 vaccination circulates in the plasma as early as one day after vaccination. Interaction with full-length S2 spike, its subunits (S1, S2), and/or peptide fragments with host molecules result in prolonged symptoms.
Recently, a subset of non-classical monocytes has been shown to harbor S2 spike protein in patients with PVS18.
Further, biodistribution studies on mRNA–LNP platforms in animal models indicate its ability to cross the blood-brain barrier, and the local S2 expression results in neurocognitive symptoms.
#3 Vaccine-induced VAIDS immune responses are triggering the stimulation of autoreactive lymphocytes.