Category: Vaccines

For those of us who did not study statistics, ‘standard deviation’ measures how spread out data is from the average. So seven standard deviations is huge!

A massive new study suggests that far fewer long-term conditions can be attributed to COVID-19 when researchers compare PCR-positive and PCR-negative patients. They are dismissing so much.
Walter M Chesnut

Sometimes something is published (more often so, these days) that is so seriously wrong that it must be addressed. No matter how ridiculous it is. A newly published study in the Proceedings of the National Academy of Sciences has generated considerable attention by reporting a 98.2% reduction in the number of clinical outcomes statistically attributable to COVID-19 when researchers changed how they selected their control population. This is one such seriously wrong, ridiculous example.

The study, Revised estimates of types and duration of long COVID symptoms based on claims records from 245 million US patients, analyzed an extraordinary 14.4 billion insurance claims from approximately 245 million Americans. The investigators compared patients who tested positive for SARS-CoV-2 with patients who tested negative, rather than relying on broader populations that included people who had never been tested.

The results were dramatic.

Between 120 and 359 days after testing, the number of clinical outcomes statistically attributed to COVID-19 declined from 286 to just five.

A 98.2% reduction.

There is a crucial distinction we must acknowledge here: this was not a 98.2% reduction in the number of people suffering from Long COVID. It was a reduction in the number of outcome categories that met the researchers’ criteria for attribution to COVID-19.

The “authors” of the study are IGNORING (intentionally, they can’t be this stupid) some extraordinarily important FACTS.

Some of the people who tested negative had almost certainly already developed Long COVID from an earlier infection that was never documented. Some of the people who tested negative almost certainly had been given Spike mRNA/injections.

A Negative PCR Test Is Not a Negative Infection/Exposure History

The central premise of the researchers’ approach is absurd. People who undergo COVID testing may differ from people who never seek testing. They may have underlying medical conditions, more frequent healthcare encounters, or other reasons for receiving medical attention. They also may prefer apples to oranges…

All a negative PCR test establishes is that SARS-CoV-2 was not detected in that particular sample at that particular time. It does not establish that the individual had never been infected. Or that they had never been exposed to the Spike Protein via mRNA/vaccination.

Most importantly, it certainly does not establish that an earlier infection/exposure had not initiated a chronic pathological process. Indeed, the National Academies’ 2024 definition of Long COVID explicitly recognizes that the condition can follow an infection that was never clinically recognized or laboratory-confirmed. The CDC likewise states that a positive SARS-CoV-2 test is not required for a Long COVID diagnosis.

A person can have Long COVID without ever having had a documented positive SARS-CoV-2 test.

This is not a controversial proposition. It is part of the accepted clinical understanding of the condition.

From the CDC itself:

Long COVID is not one illness. There is no approved laboratory test that can determine if your symptoms or conditions are due to Long COVID. A positive SARS-CoV-2 test is not required for a Long COVID diagnosis.

Long COVID Basics
https://www.cdc.gov/long-covid/about/index.html

What If the Macrophage Was Already Smoldering?

This is where my Smoldering Macrophage Syndrome (SMS) hypothesis becomes relevant.

I have proposed that SARS-CoV-2 infection/Spike exposure may initiate persistent, tissue-localized macrophage activation that continues long after the acute infection has resolved or Spike exposure occurred. In this model, the initiating infection/exposure and subsequent disease process are not necessarily simultaneous.

The virus may no longer be detectable in the upper respiratory tract. Circulating inflammatory markers may be unremarkable. Yet macrophages residing in tissues such as the lungs, heart, brain, gut, and microvasculature could remain abnormally activated, contributing to persistent inflammatory signaling, endothelial dysfunction, and tissue remodeling.

This remains a hypothesis requiring direct validation. But consider what it would mean for the interpretation of this enormous epidemiological study. Imagine an individual who experienced an undocumented SARS-CoV-2 infection months earlier, or Spike Protein exposure.

That individual could now enter the study’s comparison population despite already having a post-COVID/Spike Exposure condition.

If this occurred frequently enough, the supposedly uninfected comparison group could contain people with the very disease processes the researchers were attempting to attribute to COVID-19.

I am absolutely stunned that this nonsense was published. (Or am I?) And yes, it’s dangerous. It not only is inaccurate about the potential prevalence of Long COVID, it also indirectly seeks to dismiss the suffering of millions who are dealing with this horrific disease state. The “study” should be immediately retracted and with formal apologies from the publisher. We will not tolerate such manipulation of data or dismissal of the anguish of millions of individuals.

The system acts to protect the system and those in charge, not the public.

Increasing your own knowledge, competence and productivity, expanding your awareness and your continued vigilance are your best first line of personal defence against a system designed and managed to protect itself, not you.

“The highest activity a human being can attain is learning for understanding, because to understand is to be free.” – Spinoza, Dutch Philosopher (1632 – 1677)

Helping others to also increase their awareness, knowledge, competence and production is the next level in replacing the present system with a more equitable one.

Vaclav Havel was a Soviet dissident who wrote the book, “Power of the Powerless”. He went on to become the president of the Czech Republic.

He said, “The most powerful weapon against the state is parallel construction. More powerful that combat, protest, or anything else.”

I have seen similar written elsewhere by Buckminster Fuller:
“You never change things by fighting the existing reality.

To change something, build a new model that makes the existing model obsolete.”

So step three is to get busy building a new model that makes the existing system obsolete.

For many years I have had a saying, “It is hard for a person to rise to a higher level of awareness when their attention is being dragged down to a sick or malnourished body.”

If you would like some assistance on step 1 or 2 or 3, please feel free to reach out to me. I have not spent a large portion of my life and income on improving myself to take it to the grave without sharing!

Pfizer doesn’t want you to know this. This information doesn’t appear in any of their ads for the COVID vaccine for some reason. Baffling for sure!
Executive summary

The easiest way to tell if you are talking to someone who is intellectually honest about the COVID vaccines is to ask them for their assessment of the Levi paper.

If they claim, like UPenn Professor Jeffrey Morris does, that there is nothing to see here, then you are dealing with someone who is intellectual dishonest. The AI analysis of Morris’s attack on the Levi paper is absolutely devastating.
What the paper found

37% higher non-COVID mortality in people who got Pfizer.

That’s a train wreck.

There is no other way to spin this.

Possible explanations for the results:

The Pfizer/Moderna groups weren’t really matched

Moderna reduces non-COVID all-cause mortality by 37%

The problems:

They were matched. Barbra Dickerman wrote 2 papers on COVID vaccines and VA data showing that crude demographic matching creates extremely good morbidity matching to nobody’s surprise. But Levi’s matching was MUCH stronger than what Dickerman did (more attributes and tighter matches). And he confirmed the matching by looking at the single most devastating statistic: prior cardiovascular hospitalization rates were matched. This is not a random marker — it’s arguably the single most mortality-relevant clinical signal you can extract from administrative data. So we have no reason to believe the groups weren’t matched and every reason to believe they were.

There is no mechanism of action that could possibly explain how a COVID vaccine can reduce non-COVID all-cause mortality by 37%. If that were true, this would be the most amazing drug ever created. Moderna would be marketing their COVID vaccine as a fountain of youth. Everyone would be taking it. We’d have seen dramatic reduction in all cause mortality all over the world after rolling out the drug. No such data exists. If you plot cumulative COVID deaths over time for the world, the rollout of the vaccine didn’t change the slope one bit which means the COVID shots didn’t really save any lives. But people who opt to taking vaccines have lower mortality in general, but it is NOT caused by vaccines; it is 100% selection bias. People who opt for vaccines tend to have better access to healthcare.

The full discussion is here and it’s worth reading.
The bottom line

No two ways about it. The COVID vaccines were deadly.

They should be taken off the market.

It is absolutely appalling that businesses and schools forced people to take a vaccine that can increase mortality by 37%. But nobody is willing to talk about it, and no business has formally apologized for their mistake.

Those who want to talk about, like Professor Morris, cherry pick the evidence to support their point of view. These people are extremely dangerous. The AI analysis of Morris’s attack on the Levi paper is absolutely devastating.
Where is the outrage?

Every member of Congress should be shocked by this paper. They should be calling for replication of this study in all 50 states.

Don’t they want to know that they were responsible for over 500,000 American deaths?

Can you name one member of Congress doing that? With 535 members, you’d think that there would be at least one honest member speaking out asking for similar studies to be done, wouldn’t you?

Not one.

They don’t want to know.

Summary

The Levi paper is clear: 37% higher mortality if you got Pfizer.

None of the “explanations” can explain this difference.

Nobody is calling for more studies to replicate the Levi study.

And the Levi study was rejected by around 12 journals before being published showing you just how corrupt “science” is.

https://open.substack.com/pub/stevekirsch/p/levi-paper-showing-pfizer-is-a-toxic

(Tom: A great article giving a perspective on the last 6 years and why no accountability is developing.)

TWO QUOTES TO SET THE FRAME

The two quotes:

The first, from Robert Hunter, the bard of the Grateful Dead. And this quote may reflect something of my misspent youth: What a long, strange trip it’s been.

The second, widely attributed and of uncertain origin. We certainly live in interesting times.

https://open.substack.com/pub/rwmalonemd/p/notes-on-my-brownstone-annual-conference

BREAKING: New Zealand whistleblower Barry Young: “They RAIDED my home and ARRESTED me for exposing deadly COVID vaccine batches.”

Catastrophic death rate PFIZER batches:
Batch 1: 152 deaths out of 711 vaccinated 21.4% dead.
Batch 2: 38 deaths out of 221 17.2% dead.
Batch 3: 48 deaths out of 310 15.5% dead.

Normal background death rate in that period? Only 0.75%.

The statistical odds of this happening by chance? Roughly 1 in 100 BILLION.

His direct conclusion: “Statistically, there is no way this vaccine ISN’T killing people.”

Yet instead of launching an investigation… authorities raided his home and arrested him.

Please read and share.

(Definition: Microangiopathy (also known as microvascular disease, small vessel disease (SVD) or microvascular dysfunction) is a disease of the microvessels, small blood vessels in the microcirculation.

Small vessel diseases (SVDs) affect primarily organs that receive significant portions of cardiac output such as the brain, the kidney, and the retina. Thus, SVDs are a major etiologic cause in debilitating conditions such as renal failure, blindness, strokes from brain clots, and dementia.)

Executive Summary:
…repeated exposure to the Spike Protein causes small blood vessels to be scarred and damaged resulting in fibrosis.

It is being so injured that it is repairing repairs. Layer upon layer of non-functioning fibrous tissue.

Why is this so important? Perhaps the most urgent reason:
Microvasculature collagen I deposition was only evident in fatal COVID-19, long COVID, and in the mice exposed to the S1 subunit for 1.5 months.

Please reread that carefully. Several times, if need be. The deposition of collagen was evident not only in those who died or had Long COVID, but ALSO IN THOSE MICE WHO WERE EXPOSED TO THE SPIKE PROTEIN FOR 1.5 MONTHS.

What does this tell us?

This tells us that the Spike Protein may damage us EVEN IF IT DOES NOT CAUSE APPARENT DISEASE. Therefore, you could seem to be completely healthy, without experiencing a case of severe COVID or Long COVID, yet the Spike Protein, if present (repeatedly being injected/produced), may be slowly erasing your microvasculature. This could result in a whole host of deleterious conditions, including sudden cardiac death. And have we seen sudden cardiac death, unfortunately.

Of course, the list goes on, and virtually every major organ could have its functionality severely impaired over time. Yet without the appearance of any major symptoms while the devastation grows.

It was precisely this disease process that I predicted the Spike Protein would induce in the microvasculature.

Article:
Five years ago, I wrote a post wherein I warned that repeated exposure to the Spike Protein would result in a systemic microangiopathy resulting in fibrosis.

The microvasculature is being lacerated and scarred. It is being so injured that it is repairing repairs. Layer upon layer of non-functioning fibrous tissue. This unfortunately hearkens back to my hypothesis that the Spike Protein of SARS-CoV-2 is turning us into masses of living cartilage.

COVID-19 LOOKS LIKE RADIATION POISONING: NOT JUST A VASCULAR DISEASE BUT A DISEASE OF SYSTEMIC MICROANGIOPATHY FOLLOWED BY SYSTEMIC FIBROSIS

Walter M Chesnut

1) MY MOST IMPORTANT FINDING TO DATE – WHY COVID-19 LOOKS LIKE RADIATION POISONING COVID-19: NOT JUST A VASCULAR DISEASE BUT A DISEASE OF SYSTEMIC MICROANGIOPATHY FOLLOWED BY SYSTEMIC FIBROSIS, ALMOST CERTAINLY INDUCED BY THE TISSUE-DAMAGING EFFECTS OF THE SPIKE

The date of that post? 9:16 PM · Nov 29, 2021!!!

Just last week a major study was published confirming this exact hypothesis of mine. And due to the exact same mechanism – repeated exposure to the Spike Protein.

The question of whether the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) spike protein persists in angiotensin converting-enzyme 2–positive (ACE2+) endothelia in the central nervous system (CNS)/other sites and induces a prolonged microvasculature injury syndrome was addressed. People who died of COVID-19, with age-matched controls, and people with long COVID with symptoms persisting from 1 to 5 years, with controls of people who had recovered from COVID-19, were studied. Also compared were wild-type mice injected with the spike protein euthanized at day 5 versus 1.5 months. The ACE2+ microvasculature density in the CNS increased 19-fold in the mice exposed to spike protein for 1.5 months. Human fatal COVID-19 cases exhibited an ACE2+ CNS density of 13.2/3.9 versus 2.1/0.9 for the controls. Long COVID cases showed an ACE2 microvasculature density of 11.9/3.0 versus 3.5/0.9 for the people who recovered from COVID. Viral spike microvasculature density in the CNS was similar in the fatal COVID-19 cases (6.9/2.1) and mice (7.0/4.0) and in the skin of long COVID cases (6.0/1.9) and less in people who recovered from COVID-19 (0.9/0.3). Microvasculature collagen I deposition was only evident in fatal COVID-19, long COVID, and in the mice exposed to the S1 subunit for 1.5 months. Thus, SARS-CoV-2 spike (S1) protein can persist for months to years with an associated microvasculature pro-inflammatory state and a restrictive fibrosing microangiopathy, and exposure to high-dose S1 markedly increases ACE2 endothelial density.

Effects of Prolonged SARS-CoV-2 Spike Protein Exposure in Long COVID and a Mouse Model of the Disease
https://ajp.amjpathol.org/article/S0002-9440(26)00296-8/abstract

Why is this so important? Perhaps the most urgent reason:

Microvasculature collagen I deposition was only evident in fatal COVID-19, long COVID, and in the mice exposed to the S1 subunit for 1.5 months.

Please reread that carefully. Several times, if need be. The deposition of collagen was evident not only in those who died or had Long COVID, but ALSO IN THOSE MICE WHO WERE EXPOSED TO THE SPIKE PROTEIN FOR 1.5 MONTHS.

What does this tell us?

This tells us that the Spike Protein may damage us EVEN IF IT DOES NOT CAUSE APPARENT DISEASE. Precisely as I predicted it may. Therefore, you could seem to be completely healthy, without experiencing a case of severe COVID or Long COVID, yet the Spike Protein, if present (repeatedly being injected/produced), may be slowly erasing your microvasculature. This could result in a whole host of deleterious conditions, including sudden cardiac death. And have we seen sudden cardiac death, unfortunately.

The coronary vascular system has a unique structure and function that is adaptive to myocardial demand. It is composed of a continuous network of vessels receding in size from epicardial arteries to the microvascular circulation. Failure to meet myocardial demand results in ischemia, angina, and adverse myocardial outcomes. It is evident that 50 % of patients with angina have a non-obstructive coronary disease and 66 % of these patients have coronary microvascular dysfunction (CMD). The impact of CMD on the atria and ventricles is exhibited through its association with atrial fibrillation and distortion of ventricular repolarization. Ultimately, this influence increases the risk of mortality, morbidity, and sudden cardiac arrest. CMD serves as an independent risk for atrial fibrillation, increases ventricular electrical inhomogeneity, and contributes to the progression of cardiac disease. The underlying pathogenesis may be attributed to oxidative stress evident through reactive oxygen species, impaired vasoactive function, and structural disorders such as fibrotic changes. Myocardial ischemia, brought about by a demand-supply mismatch in CMD, may create a milieu for ventricular arrythmia and sudden cardiac arrest through distortion of ventricular repolarization parameters such as QT dispersion and corrected QT dispersion.

Coronary microvascular dysfunction, arrythmias, and sudden cardiac death: A literature review
https://pmc.ncbi.nlm.nih.gov/articles/PMC10998042/

Of course, the list goes on, and virtually every major organ could have its functionality severely impaired over time. Yet without the appearance of any major symptoms while the devastation grows.

We need to have studies on the microvasculature of the general population by COVID infection history, COVID vaccination history and to see if Spike is circulating in those who have neither had COVID nor a COVID vaccine. And yes, this is extremely important and urgent. Clearly, I wish my warnings had been heeded half a decade ago. We will fight on.

Finish reading: https://wmcresearch.substack.com/p/confirmation-of-my-finding-from-five