Sun is NOT The Enemy!

A massive Swedish study followed 30,000 women for over 20 years and found that those who actively sought sun exposure had dramatically lower death rates from cancer, heart disease, and all causes.

The shocking part? Sun avoiders had roughly double the overall mortality.

Even heavy smokers who got plenty of sun had similar death rates to non-smokers who avoided it.

Sunlight appears to extend life through vitamin D, nitric oxide, and immune support – yet we’re still told to hide from it. Are you getting enough sun?

https://x.com/UltraDane/status/2038691028937675004?s=20

Lifting Weights Helps Your Brain Process More Information

Barbell Weights

  • Moderate-intensity resistance workouts can sharpen thinking by improving reaction time without reducing accuracy, enhancing inhibitory control, working memory, and speeding brain-processing signals
  • Cognitive gains likely come from temporary increases in systolic blood pressure, which enhance blood flow and neural efficiency, helping the brain update information faster and coordinate attention-related networks more effectively
  • Children and teenagers also benefit from resistance training, showing small but consistent improvements in cognition, on-task classroom behavior, and academic performance, especially among youth with lower baseline muscular fitness
  • For longevity, 40 to 60 minutes of strength training weekly is optimal. Excessive lifting reduces benefits, increases strain, and even worsens mortality outcomes compared to moderate or minimal resistance exercise
  • Blood flow restriction (KAATSU) training amplifies strength gains and bone benefits using low loads. It can be incorporated into daily activities, stimulating muscle and vascular adaptations without heavy lifting

https://articles.mercola.com/sites/articles/archive/2026/03/20/resistance-training-cognitive-benefits.aspx

The COVID Vax Signal They Didn’t Follow

Why this “reassuring” vaccine study may be missing the most important risk pattern

A paper has just been published examining COVID-19 vaccination and sudden death in younger individuals. It has been widely shared because it appears reassuring. The conclusion: no evidence that COVID-19 vaccines increase the risk of sudden cardiac death in healthy young adults.

At face value, that sounds like the end of the discussion. For me, it is the beginning. When a study gives a clean answer to a complex biological problem, it is worth going back into the data to see what has been simplified.

The Finding That Should Have Been the Focus

When I looked at the baseline characteristics, one detail immediately stood out. Individuals who had a recent COVID-19 infection within 90 days were more than twice as likely to be in the group that died compared to those who survived.

This is not a subtle difference. This is a strong signal. And yet, it is not the headline.

Instead, it is adjusted for, controlled, and moved into the background so that the primary question — whether vaccination alone is associated with sudden death — can be answered. But that approach assumes something I do not believe reflects reality.

Abdel-Qadir, Husam, et al. “Association between COVID-19 vaccination and sudden death in apparently healthy younger individuals: A population-based case-control study.” PLoS medicine 23.3 (2026): e1004924.

The study treats vaccination and infection as separate variables. Statistically, that is standard. Biologically, it is incomplete.

We are no longer dealing with a population that has experienced a single exposure. We are dealing with individuals who have been immune primed through vaccination or prior infection, and then repeatedly exposed to the virus. The relevant question is not whether vaccination or infection independently increases risk. It is what happens when they occur in sequence.

The COVID Storm

This is what I have been describing for some time as a “COVID storm.” A subgroup of individuals who have experienced immune priming followed by further infection. In that context, the immune response may not behave in a predictable or balanced way. It may become dysregulated. In the heart, this could manifest as inflammation, altered metabolic function, or electrical instability — and in some individuals, that may translate into clinically significant events.

A Pattern That Doesn’t Sit Comfortably

There is another signal in the study that reinforces this concern. Individuals who received only one dose of vaccination appear less “protected” than those who received multiple doses. That is not a straightforward biological gradient. It is a divergence. And divergence usually means the groups are not the same.

Some individuals continue with further doses. Others stop. In clinical medicine, when someone stops after an initial exposure, it is rarely random. It often reflects intolerance, early symptoms, or a different underlying physiology.

The Question That Wasn’t Asked

What I would have wanted to see in this study is simple. Of the individuals who had a recent infection and then died, what proportion were vaccinated? How many doses had they received? What was the time interval between their last exposure and infection?

That is where the answer is likely to be found. If there is a higher-risk subgroup, it will not sit neatly in “vaccinated” or “unvaccinated.” It will sit in the interaction between exposure and response over time.

We are seeing rising patterns across multiple cardiovascular conditions since 2020 — arrhythmias, heart failure, thrombotic disease, inflammatory cardiac conditions. This is not confined to one diagnosis.

To dismiss these patterns without fully interrogating the underlying mechanisms is not good enough. This is not about ideology. It is not about being pro or anti any intervention. It is about understanding risk properly.

A Lesson From History

I often think about how long it took for the link between smoking and disease to be fully accepted. There were studies that created doubt, arguments about confounding, calls for more data. For decades, uncertainty was enough to delay clarity.

That does not mean the conclusions today are wrong. But it does mean we should be cautious about assuming we already have the full picture.

The conclusion of this study may well be correct in its narrow framing. Vaccination alone may not increase the risk of sudden cardiac death in healthy young individuals. But that is not the full question.

The more important question is this: what is the risk in individuals who are immune primed and then experience a recent infection?

Until that is answered, we are simplifying a complex biological system into variables that are easier to analyse — but not necessarily accurate to reality.

Final Thought

This has never been about proving that one factor is responsible. It has always been about recognising that we may be dealing with a new pattern of disease — one that emerges not from a single exposure, but from the interaction of exposures over time.

If we continue to analyse these events in isolation, we will miss it. And if we miss it, we cannot manage it.

That is the risk we should be paying attention to.

https://open.substack.com/pub/philipmcmillan/p/the-covid-vax-signal-they-didnt-follow

Nolan Ryan

Nolan Ryan

In 1991, a 44-year-old pitcher with a stress fracture in his lower back, a throbbing heel, and a body that felt every one of his 27 major-league seasons stepped to the mound on four days’ rest—because it was Arlington Appreciation Night, and he refused to disappoint the fans who had stuck with him.

Nolan Ryan didn’t expect to finish the game.

He had told his pitching coach, Tom House, and manager Bobby Valentine before the start: “My back hurts, my heel hurts, I’ve been pounding Advil all day. I don’t feel good. I feel old today. Watch me closely.”

Valentine alerted the umpires that an early pitching change was likely. Someone was already warming up in the bullpen.

Then Ryan threw his first pitch.

Ninety-four miles per hour.

The second pitch: ninety-five.

Batters who weren’t even born when Ryan made his major-league debut with the New York Mets in 1966 started swinging helplessly at fastballs they never saw coming. Major leaguers looked like Little Leaguers. By the second inning, his curveball was dropping off the table like a trapdoor opening beneath their feet. He struck out the side on called strikes—pitches so perfect the batters didn’t even bother arguing. They just turned and walked back to the dugout in silent disbelief.

The Texas Rangers infielders jogged off the field, exchanged glances, and grinned. They could feel it. Something special was happening.

By the sixth inning, Arlington Stadium was filling beyond capacity. The official attendance was 33,439, but it felt like 50,000. Word had spread throughout the Dallas-Fort Worth area. People abandoned their Wednesday night plans and rushed to the ballpark. History was unfolding in real time.

Nolan Ryan—the man who could barely stand upright three hours earlier—was throwing a no-hitter against the best-hitting team in baseball.

The ninth inning arrived. Future Hall of Famer Roberto Alomar stepped to the plate. Two decades earlier, Roberto’s father Sandy had been Ryan’s teammate with the California Angels. Little Roberto used to shag fly balls and play catch with Nolan before games.

Now, twenty years later, that same kid stood between Ryan and immortality.

The count went to 2-2. Ryan wound up and fired a fastball.

Alomar swung.

Missed.

Strike three.

Nolan Ryan had just thrown the seventh no-hitter of his career—three more than anyone in baseball history. At 44 years and 90 days old, he became the oldest pitcher ever to accomplish the feat.

The final line: seven innings of hitless baseball, 16 strikeouts, 122 pitches thrown. He did it on four days’ rest, with a stress fracture in his lower back, against a Toronto lineup that would go on to win the AL East.

When reporters crowded around his locker afterward, Ryan didn’t talk about records or statistics. His answer was simple and genuine: “It was the most rewarding no-hitter of them all because it came in front of my fans on Arlington Appreciation Night. My career is complete now. I got one for the fans in Arlington.”

Nolan Ryan pitched for 27 seasons in the major leagues. Seven different presidents occupied the White House during his career. He struck out players from four different decades—everyone from Roger Maris in the 1960s to Mark McGwire in the 1990s.

He retired with 5,714 career strikeouts (a record that still stands), 324 wins, and those seven no-hitters. Twenty-three years later, no one has come remotely close to any of those marks.

Modern baseball is obsessed with pitch counts and load management. Teams monitor every throw with sophisticated tracking technology. Innings are carefully restricted. Young arms are bubble-wrapped and protected.

Nolan Ryan threw nearly 5,000 innings over two decades before that seventh no-hitter.

He never got the memo.

There will never be another Nolan Ryan.

And on that May night in Arlington, when a broken-down 44-year-old refused to accept what his body was telling him, we witnessed something we’ll never see again.

Sometimes the greatest performances come when you have every reason to fail—and choose greatness anyway.

Gordon Cooper

Gordon Cooper

On May 16, 1963, Gordon Cooper was alone in Faith 7, orbiting Earth at 17,500 miles per hour in a capsule so small he could barely turn around.

He had been in space for more than thirty-four hours.

Then the alarms began.

First a faulty sensor falsely indicated the spacecraft was tumbling out of control. Cooper calmly switched it off. Then came the real emergency: a short circuit knocked out the entire automatic attitude-control system—the system that kept the capsule properly oriented for reentry. Without it, the spacecraft could not be aligned for the precise angle needed to survive the plunge back into the atmosphere.

Too shallow, and it would skip off the air like a stone across water, back into orbit. Too steep, and it would burn up like a meteor. The window was narrow. Every computer that was supposed to make that calculation was dead.

On the ground, Mission Control watched the telemetry go dark. They could see the problem. They could not fix it.

Cooper did not panic.

He uncapped a grease pencil and drew reference lines directly on the inside of his window to track the horizon against the stars. He had spent months memorizing star patterns as part of backup navigation training. Now he used them. He aligned the capsule manually by eye, matching the horizon marks to known constellations.

He timed the reentry burn with his wristwatch.

When the moment arrived—calculated in his head, confirmed by the stars—he fired the retrorockets. The capsule shuddered. The sky turned to plasma. For several minutes, radio blackout swallowed him whole. No voice from Earth could reach him. No data came back.

Then the parachutes deployed.

Faith 7 splashed down in the Pacific, four miles from the recovery ship USS Kearsarge—the most accurate splashdown of the entire Mercury program.

A man with a grease pencil, a wristwatch, and starlight had outperformed every automated system NASA had built.

We often speak of technology as the hero of spaceflight. And it frequently is.

But Gordon Cooper’s flight is a reminder that behind every machine, there must still be a human being who can look out the window, think clearly under crushing pressure, and decide what to do when everything else fails.

The final backup was never the software.

It was him.

Avivo Village

Avivo Village

Avivo Village in Minneapolis is an innovative shelter designed to provide people experiencing homelessness with a safer and more stable place to stay. Instead of large dorm-style spaces typical of many shelters, it offers small, private, lockable rooms inside a warehouse, giving residents greater dignity, security, and personal space.

Beyond providing warmth and safety during harsh winters, the community also connects residents with essential services such as mental health care, addiction treatment, and housing support. By combining shelter with comprehensive assistance, Avivo Village aims to help people move beyond temporary emergency housing and toward long-term stability.