Author: TomsWrite

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Alexey and the Routers

Alexey and the Routers

In 2018, MikroTik, a Latvian maker of networking gear, was dealing with a serious security flaw in its routers. A fix existed, but tens of thousands of devices around the world were still running old software, and criminal botnets were scanning the internet for them.

Once inside, the attackers put the routers to work mining cryptocurrency and joining coordinated attacks, often without the owners having any idea. A security expert who used the name Alexey watched this happen and decided the owners weren’t going to patch the problem fast enough on their own.

So he wrote an automated script that went looking for the same vulnerable routers, logged into them through the same weakness and changed each one’s firewall rules to block the ports the attackers were using. He didn’t look at anyone’s data, harvest passwords or install software of his own. Before leaving each device he left a brief note explaining the change and urging the owner to update.

Alexey said his only goal was to protect ordinary internet users, but what he’d done was still unauthorized access to other people’s equipment. The episode split the security world, with some praising him as a digital vigilante and others arguing that breaking into someone’s device without permission is wrong whatever the motive.

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Confirmation of My Finding from Five Years Ago: Repeated Spike Exposure Induces a Restrictive Fibrosing Microangiopathy

Please read and share.

(Definition: Microangiopathy (also known as microvascular disease, small vessel disease (SVD) or microvascular dysfunction) is a disease of the microvessels, small blood vessels in the microcirculation.

Small vessel diseases (SVDs) affect primarily organs that receive significant portions of cardiac output such as the brain, the kidney, and the retina. Thus, SVDs are a major etiologic cause in debilitating conditions such as renal failure, blindness, strokes from brain clots, and dementia.)

Executive Summary:
…repeated exposure to the Spike Protein causes small blood vessels to be scarred and damaged resulting in fibrosis.

It is being so injured that it is repairing repairs. Layer upon layer of non-functioning fibrous tissue.

Why is this so important? Perhaps the most urgent reason:
Microvasculature collagen I deposition was only evident in fatal COVID-19, long COVID, and in the mice exposed to the S1 subunit for 1.5 months.

Please reread that carefully. Several times, if need be. The deposition of collagen was evident not only in those who died or had Long COVID, but ALSO IN THOSE MICE WHO WERE EXPOSED TO THE SPIKE PROTEIN FOR 1.5 MONTHS.

What does this tell us?

This tells us that the Spike Protein may damage us EVEN IF IT DOES NOT CAUSE APPARENT DISEASE. Therefore, you could seem to be completely healthy, without experiencing a case of severe COVID or Long COVID, yet the Spike Protein, if present (repeatedly being injected/produced), may be slowly erasing your microvasculature. This could result in a whole host of deleterious conditions, including sudden cardiac death. And have we seen sudden cardiac death, unfortunately.

Of course, the list goes on, and virtually every major organ could have its functionality severely impaired over time. Yet without the appearance of any major symptoms while the devastation grows.

It was precisely this disease process that I predicted the Spike Protein would induce in the microvasculature.

Article:
Five years ago, I wrote a post wherein I warned that repeated exposure to the Spike Protein would result in a systemic microangiopathy resulting in fibrosis.

The microvasculature is being lacerated and scarred. It is being so injured that it is repairing repairs. Layer upon layer of non-functioning fibrous tissue. This unfortunately hearkens back to my hypothesis that the Spike Protein of SARS-CoV-2 is turning us into masses of living cartilage.

COVID-19 LOOKS LIKE RADIATION POISONING: NOT JUST A VASCULAR DISEASE BUT A DISEASE OF SYSTEMIC MICROANGIOPATHY FOLLOWED BY SYSTEMIC FIBROSIS

Walter M Chesnut

1) MY MOST IMPORTANT FINDING TO DATE – WHY COVID-19 LOOKS LIKE RADIATION POISONING COVID-19: NOT JUST A VASCULAR DISEASE BUT A DISEASE OF SYSTEMIC MICROANGIOPATHY FOLLOWED BY SYSTEMIC FIBROSIS, ALMOST CERTAINLY INDUCED BY THE TISSUE-DAMAGING EFFECTS OF THE SPIKE

The date of that post? 9:16 PM · Nov 29, 2021!!!

Just last week a major study was published confirming this exact hypothesis of mine. And due to the exact same mechanism – repeated exposure to the Spike Protein.

The question of whether the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) spike protein persists in angiotensin converting-enzyme 2–positive (ACE2+) endothelia in the central nervous system (CNS)/other sites and induces a prolonged microvasculature injury syndrome was addressed. People who died of COVID-19, with age-matched controls, and people with long COVID with symptoms persisting from 1 to 5 years, with controls of people who had recovered from COVID-19, were studied. Also compared were wild-type mice injected with the spike protein euthanized at day 5 versus 1.5 months. The ACE2+ microvasculature density in the CNS increased 19-fold in the mice exposed to spike protein for 1.5 months. Human fatal COVID-19 cases exhibited an ACE2+ CNS density of 13.2/3.9 versus 2.1/0.9 for the controls. Long COVID cases showed an ACE2 microvasculature density of 11.9/3.0 versus 3.5/0.9 for the people who recovered from COVID. Viral spike microvasculature density in the CNS was similar in the fatal COVID-19 cases (6.9/2.1) and mice (7.0/4.0) and in the skin of long COVID cases (6.0/1.9) and less in people who recovered from COVID-19 (0.9/0.3). Microvasculature collagen I deposition was only evident in fatal COVID-19, long COVID, and in the mice exposed to the S1 subunit for 1.5 months. Thus, SARS-CoV-2 spike (S1) protein can persist for months to years with an associated microvasculature pro-inflammatory state and a restrictive fibrosing microangiopathy, and exposure to high-dose S1 markedly increases ACE2 endothelial density.

Effects of Prolonged SARS-CoV-2 Spike Protein Exposure in Long COVID and a Mouse Model of the Disease
https://ajp.amjpathol.org/article/S0002-9440(26)00296-8/abstract

Why is this so important? Perhaps the most urgent reason:

Microvasculature collagen I deposition was only evident in fatal COVID-19, long COVID, and in the mice exposed to the S1 subunit for 1.5 months.

Please reread that carefully. Several times, if need be. The deposition of collagen was evident not only in those who died or had Long COVID, but ALSO IN THOSE MICE WHO WERE EXPOSED TO THE SPIKE PROTEIN FOR 1.5 MONTHS.

What does this tell us?

This tells us that the Spike Protein may damage us EVEN IF IT DOES NOT CAUSE APPARENT DISEASE. Precisely as I predicted it may. Therefore, you could seem to be completely healthy, without experiencing a case of severe COVID or Long COVID, yet the Spike Protein, if present (repeatedly being injected/produced), may be slowly erasing your microvasculature. This could result in a whole host of deleterious conditions, including sudden cardiac death. And have we seen sudden cardiac death, unfortunately.

The coronary vascular system has a unique structure and function that is adaptive to myocardial demand. It is composed of a continuous network of vessels receding in size from epicardial arteries to the microvascular circulation. Failure to meet myocardial demand results in ischemia, angina, and adverse myocardial outcomes. It is evident that 50 % of patients with angina have a non-obstructive coronary disease and 66 % of these patients have coronary microvascular dysfunction (CMD). The impact of CMD on the atria and ventricles is exhibited through its association with atrial fibrillation and distortion of ventricular repolarization. Ultimately, this influence increases the risk of mortality, morbidity, and sudden cardiac arrest. CMD serves as an independent risk for atrial fibrillation, increases ventricular electrical inhomogeneity, and contributes to the progression of cardiac disease. The underlying pathogenesis may be attributed to oxidative stress evident through reactive oxygen species, impaired vasoactive function, and structural disorders such as fibrotic changes. Myocardial ischemia, brought about by a demand-supply mismatch in CMD, may create a milieu for ventricular arrythmia and sudden cardiac arrest through distortion of ventricular repolarization parameters such as QT dispersion and corrected QT dispersion.

Coronary microvascular dysfunction, arrythmias, and sudden cardiac death: A literature review
https://pmc.ncbi.nlm.nih.gov/articles/PMC10998042/

Of course, the list goes on, and virtually every major organ could have its functionality severely impaired over time. Yet without the appearance of any major symptoms while the devastation grows.

We need to have studies on the microvasculature of the general population by COVID infection history, COVID vaccination history and to see if Spike is circulating in those who have neither had COVID nor a COVID vaccine. And yes, this is extremely important and urgent. Clearly, I wish my warnings had been heeded half a decade ago. We will fight on.

Finish reading: https://wmcresearch.substack.com/p/confirmation-of-my-finding-from-five

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A new AI-driven medical danger

Critical skills testing is under threat
Alex Berenson
Nov 2
(Note: the email that’s posted below arrived over the weekend. I am keeping the author anonymous, per her request. But the problem of artifical-intelligence-powered cheating on tests she highlights is real and growing. For jobs like nursing, this is not just an academic problem. It’s a threat to patient safety.
Companies and boards that administer critical licensing tests probably need to end any remote, at-home exams. Instead, they should offer tests only at centralized proctored locations on laptops without ANY Internet connections that they control and store themselves. Even then they face the risk of AI-powered glasses, though those should be more easily detectable.
It’s sad that we’ve reached this pass, but the reality is what it is.)
Hi Alex,
I’m glad you are covering healthcare right now….keep going with that, please!
You have also talked about AI in general and cheating in higher education. I am a nurse educator, a Clinical Assistant Professor at a well-respected university in the XXX. Note the “clinical” in my job title – it means I do not have tenure. That means I have to watch what I do and say.
I just became aware of a new way that our nursing students have found to cheat. Our students take their exams on a computer that uses a lockdown browser to make sure they cannot access their computer or the internet as a way to cheat during the exam.
[Note: a lockdown browser is essentially what it sounds like — it bars a student from any connection to the Internet or opening other tabs or programs on a computer while she’s taking a test. Some lockdown browsers include software (yes, inevitably, it’s AI-powered) designed to catch students taking tests remotely who use a second computer or their phones to connect to AI engines.]
Well, we have now learned that students can use AI to get past that lockdown. A colleague tested it out. She was able to ask ChatGPT to set up an “overlay” on her -computer. (I don’t know the technical terms or how it works at that level).
She then went into the lockdown browser, and while taking an exam, she could highlight the text of the question and then, through this overlay, ChatGPT would send her a message that would appear on the computer screen with the answer to the question. This essentially means the student doesn’t have to know the content at all. In nursing education, we have always counted on the fact that even if a student graduates by cheating, they would never make it to be a practicing RN because they still have to pass the NCLEX [National Council Licensure Examination, the basic professional nursing test] to get their license.
Well… guess what? The NCSBN [National Council of State Boards of Nursing] (they make the NCLEX exam) is actively planning for nursing graduates to be able to take their exams at home. This would likely mean they would be using a lockdown browser and remote proctor of some sort. This is frightening to me.
The pace of AI being used for cheating is faster than the pace of developing ways to combat it. Many faculty where I work are switching to paper exams to avoid this type of cheating. That mode of testing is time-consuming for faculty, so I doubt that everyone will care enough to do it. I also doubt my school would require it.
I am hoping someone like you can get the word out about this. In addition to the total mess that is going on with the regulation and cost of healthcare, we are about to have nurses (and probably doctors) enter the healthcare workforce after cheating their way through school. I am telling my family to stay as healthy as possible so you don’t have to trust your life to an unknown nurse or doctor.
Feel free to use this information as you see fit, but please keep my name and location confidential. I cannot afford to get fired.
Thanks,
XXX
(August’s piece about AI and cheating in higher education)
On diversity, equity, AI cheating, and the cynicism destroying higher education (from students, professors, and administrators alike)
Alex Berenson
© 2026 Alex Berenson
548 Market Street PMB 72296, San Francisco, CA 94104
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Two Simple Food Hacks That May Help Ease Inflammation and Pain

Glycine In Water

Glycine, a simple amino acid, and cutting linoleic acid (LA) to less than 2% of your daily calories share an overlapping list of reported benefits, including less inflammation and pain, with plausible links to fewer migraines and better tolerance to the sun.

In animal studies, glycine opens a chloride channel on immune cells that blocks the calcium surge they need to launch an inflammatory attack. Oxidized LA byproducts do the opposite, triggering calcium signals in pain nerves and immune cells. The same calcium-blocking mechanism may also help protect against electromagnetic fields from wireless devices.

Research in mice suggests that a glycine receptor in the spinal cord works as a brake on pain. Prostaglandin E2, made from arachidonic acid, which your body makes from LA, can switch that brake off.

Two recent animal studies also suggest LA can drive a destructive type of cell death in joint cartilage, and glycine can block that type of cell death in rat cartilage.

A randomized trial that cut LA to 1.8% of calories or less, while adding omega-3 fats, reduced headache days by four a month, twice the benefit of adding omega-3s alone.

Your body makes only about 3 grams of glycine a day and may fall roughly 10 grams short of what it needs. The two hacks: take about 10 grams of glycine daily, and keep LA under 2% of your calories.

Finish reading:  https://articles.mercola.com/sites/articles/archive/2026/11/01/glycine-linoleic-acid-inflammation-pain.aspx

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Three Days of Darkness

Three Days of Darkness

Nation First looks into the unsettling resemblance between modern emergency checklists and an old Catholic prophecy.

Britain wants three days of water in every cupboard: three litres a person a day, three days of food, a torch, a wind-up radio. Japan’s disaster minister asks for three. France’s Normandy prefecture has published a 72-hour kit list. Belgium, the Netherlands, Latvia, Lithuania and Ireland say 72 hours. The European Commission has made 72-hour household self-sufficiency an official goal, and 33 governments now sit on a map of the advice.

Planners call the number practical. It is the window before organised help can be expected to arrive.

It is also the length of a Catholic prophecy two centuries old.

Governments are telling households to prepare for three days of disruption, echoing an old Catholic warning.

The Three Days of Darkness prophecy describes a world plunged into darkness, with the faithful sheltering indoors.

The resemblance invites two readings: independent practical planning or the exploitation of an inherited religious story.

A familiar script could encourage obedience and lead people to interpret a blackout as fulfilment, leaving its causes unquestioned.

The mystics promise renewal, while governments offer a timetable that depends on their ability to deliver.

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Quote of the Day

“A quarrel is quickly settled when deserted by one party; there is no battle unless there be two.”
Lucius Annaeus Seneca – Philosopher, Statesman, Dramatist (5 BC – 65 AD)

Just because someone invited you to an argument doesn’t mean you have to attend.

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The Forgotten Causes of High Blood Pressure & How to Treat It

Elastic vs Stiff Arteries

The Great Blood Pressure Scam, the Dangers of Blood Pressure Medications, and the Forgotten Research That Shows How to Restore Circulatory Health

Story at a Glance:

•Elevated blood pressure is the most common chronic disease, and as the decades go by more and more people are declared hypertensive.

•Remarkably, at least 25% of all hypertension diagnoses are due to inaccurate measurements, and there is still no known reason for why over 90% of people are hypertensive.

•Aggressively treating everyone’s blood pressure is justified under the belief it prevents cardiovascular disease. However, in most cases it has never been proven to reduce heart disease—rather it only leads to a small reduction in strokes (hence why these medications were rebranded to treat “cardiovascular disease”). Instead, we keep on putting more Americans on these medications (with the most recent 2025 update making 26.8 million low-risk Americans potential candidates for blood pressure medications).

•Many of the misunderstandings with heart disease arise from the fact that impaired circulation or damage to the blood vessels will cause blood pressure to go up and their correlation being misinterpreted to instead believe high blood pressure causes cardiovascular disease.

•A vast body forgotten Russian research corroborated what a few Western pioneers discovered by directly measured what happens to blood pressure when you change the thickness of the blood itself, and found the circulatory impairment comes first, that lowering blood pressure does nothing to correct it, and that several widely used blood pressure medications make it worse.

•In this article, I will discuss the actual causes of high blood pressure, the dangers of commonly used blood pressure medications, the safest pharmaceutical and natural ways to reduce blood pressure directly, and our preferred methods for treating the underlying causes of high blood pressure.

Ever since I first encountered the medical field, something struck me as off about their relentless focus on blood pressure. Before long, I began to notice that the blood pressures the same acquaintances (e.g., relatives or friends) shared with me varied immensely. As I was pondering this, a long-time spiritual teacher shared with me their belief that the relentless focus on blood pressure was due to it being much easier to measure than blood perfusion (healthy blood flow).

Then, as I became more acquainted with the medical field, I began to notice a consistent pattern—whenever a drug existed that could treat a number or statistic, as the years went by, the acceptable number kept on being narrowed, making more and more people eligible to take the drugs that treated the number.

For example, as I discussed recently, once the statins drugs entered the market (which unlike their predecessors, could effectively lower cholesterol), the acceptable blood cholesterol levels kept on being lowered, and before long almost everyone was told they would die from a heart attack unless they started a statin—despite statins have an almost non-existing mortality benefit (e.g., taking them for 5 years at best makes you live 3-4 days longer) and causing (often severe) side effects for roughly 20% of users. Broadly recommending these drugs hence appears unconscionable, but as I showed in that article, these unjustifiable guidelines were a product of clever pharmaceutical marketing and targeted bribery of public officials.

In this article, I will attempt to show how something similar happened in the field of blood pressure. As this is an immensely controversial position to take (e.g., measuring and documenting blood pressure is one of the most routine procedures during a medical visit), I’ve done my best to clearly present the evidence for this perspective so you can make your own determination.

https://www.midwesterndoctor.com/p/the-forgotten-causes-of-high-blood?r=yh8aw