![\"ATP](\"https:\/\/www.tomgrimshaw.com\/tomsblog\/wp-content\/uploads\/2026\/05\/ATP_Production_In_Mitochondria.jpg\")
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The term is exactly what it sounds like: inflammation + aging. Chronic, low-grade inflammation accelerating the aging of your cells, your tissues, and your body as a whole.<\/p>\n
Inflammation drives aging. Aging drives inflammation. The loop feeds itself.<\/p>\n
And it can be completely silent. The early signals \u2014 fatigue, stiffness, brain fog, skin losing its vitality \u2014 get waved away as normal.<\/p>\n
But over time, that quiet fire does real damage. DNA accumulates harm it can\u2019t repair. Tissues break down faster than they\u2019re rebuilt. And the diseases we associate with old age \u2014 heart disease, type 2 diabetes, Alzheimer\u2019s, osteoporosis \u2014 take root. Not because of time. Because of the inflammation those years carried with them.<\/p>\n
There\u2019s a useful reframe here. For decades, medicine has focused on cholesterol as the villain in heart disease. But cholesterol itself isn\u2019t the problem. Your body makes it deliberately \u2014 it\u2019s essential for hormones, cell membranes, and brain function.<\/p>\n
The problem is what happens to cholesterol when it meets chronic inflammation. Inflammation oxidises it. And it\u2019s the oxidised cholesterol that damages arteries and forms plaque.<\/p>\n
Research shows that when inflammation is low, cholesterol levels matter far less. When inflammation is high, even \u201cnormal\u201d cholesterol becomes dangerous.<\/p>\n
We\u2019ve been targeting the victim instead of the cause. That\u2019s the whole story of inflammaging in a nutshell.<\/p>\n
The encouraging part? Inflammatory markers like C-reactive protein and homocysteine can be measured with simple blood tests. When those markers come down \u2014 through diet, through lifestyle \u2014 the rate of cellular aging slows with them.<\/p>\n